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4) 脳卒中に伴う高血圧(シンポジウム 高血圧の病態と治療, 第437回新潟医学会)
http://hdl.handle.net/10191/41113
http://hdl.handle.net/10191/41113fc9b2ad6-46ed-4785-9d08-b3d1c0572745
| 名前 / ファイル | ライセンス | アクション |
|---|---|---|
103(1)_13-18.pdf (1.4 MB)
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| Item type | 紀要論文 / Departmental Bulletin Paper(1) | |||||
|---|---|---|---|---|---|---|
| 公開日 | 2016-04-08 | |||||
| タイトル | ||||||
| タイトル | 4) 脳卒中に伴う高血圧(シンポジウム 高血圧の病態と治療, 第437回新潟医学会) | |||||
| タイトル | ||||||
| タイトル | 4) 脳卒中に伴う高血圧(シンポジウム 高血圧の病態と治療, 第437回新潟医学会) | |||||
| 言語 | en | |||||
| 言語 | ||||||
| 言語 | jpn | |||||
| キーワード | ||||||
| 主題Scheme | Other | |||||
| 主題 | Arterial lesions in cerebrovascular diseases | |||||
| キーワード | ||||||
| 主題Scheme | Other | |||||
| 主題 | Risk factors | |||||
| キーワード | ||||||
| 主題Scheme | Other | |||||
| 主題 | Cerebral blood flow | |||||
| キーワード | ||||||
| 主題Scheme | Other | |||||
| 主題 | Management and Treatment | |||||
| キーワード | ||||||
| 主題Scheme | Other | |||||
| 主題 | Secondary prevention | |||||
| キーワード | ||||||
| 主題Scheme | Other | |||||
| 主題 | 脳卒中の血管病変 | |||||
| キーワード | ||||||
| 主題Scheme | Other | |||||
| 主題 | 危険因子 | |||||
| キーワード | ||||||
| 主題Scheme | Other | |||||
| 主題 | 脳血流 | |||||
| キーワード | ||||||
| 主題Scheme | Other | |||||
| 主題 | 治療 | |||||
| キーワード | ||||||
| 主題Scheme | Other | |||||
| 主題 | 二次予防 | |||||
| 資源タイプ | ||||||
| 資源 | http://purl.org/coar/resource_type/c_6501 | |||||
| タイプ | departmental bulletin paper | |||||
| その他のタイトル | ||||||
| その他のタイトル | Cerebrovascular Diseases and Hypertension(Hypertension : Pathophysiology and Treatment) | |||||
| 著者 |
高橋, 壮一郎
× 高橋, 壮一郎 |
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| 著者別名 | ||||||
| 識別子Scheme | WEKO | |||||
| 識別子 | 139915 | |||||
| 姓名 | Takahashi, Soichiro | |||||
| 抄録 | ||||||
| 内容記述タイプ | Abstract | |||||
| 内容記述 | The central nervous system is a major target organ for the expression of hypertensive disease. Sustained increases in systemic blood pressure (BP) may produce pathological alteration in the intracranial vasculature. The major arterial changes found in hypertensive brains are large-vessel atherosclerosis and small-vessel fibrinoid necrosis as well as arteriolosclerosis. The results of these vascular derangements appear to be parenchymal infarction, commonly encountered in hyper beta-cholesterolemic patients, and intracranial hemorrhage and small lacunar infarcts, which are almost exclusively confined to hypertensive patients. The arteries in the ischemic area lose their normal autoregulatory control, and so any reduction in BP may reduce blood flow to an already compromised part of the brain. On the other hand, a very high BP may directly cause further ischemia, as in hypertensive encephalopathy, or hemorrhage. The wisest course would seem to be that the BP should only be reduced in the acute stage when the stroke is known to be due to a hemorrhage, or when encephalopathy is suspected, and then cautiously. In the chronic stage, there is little doubt that, in the long term, active control of hypertension after a first stroke is effective in reducing the risk of stroke. Accordingly, high BP should be lowered gently to a normal level with anti-hypertensive agents such as calcium antagonists, ACE inhibitors, beta-adrenoceptor antagonists with ISA and hydralazine delivatives. | |||||
| 書誌情報 |
新潟医学会雑誌 en : 新潟医学会雑誌 巻 103, 号 1, p. 13-18, 発行日 1989-01 |
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| 出版者 | ||||||
| 出版者 | 新潟医学会 | |||||
| ISSN | ||||||
| 収録物識別子タイプ | ISSN | |||||
| 収録物識別子 | 00290440 | |||||
| 書誌レコードID | ||||||
| 収録物識別子タイプ | NCID | |||||
| 収録物識別子 | AN00182415 | |||||
| 著者版フラグ | ||||||
| 値 | publisher | |||||
