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In addition, Ly haplotypes of the former EAT-progressive mouse strains classified were Ly-1^a (except AKR) and Ly-2^a, and those of the latter EAT-regressive mouse strains displayed Ly-1^b and Ly-2^b as common features. To see how the shift restricted in H-2 or in Ly haplotype affects their susceptibility, EAT progression in H-2 congenic and in Ly congenic mice was compared with that in standard mice. The results are as follows. 1) B10(H-2^b) as a standard was EAT-regressive. B10. D2(H-2^d), which is H-2 congenic between B10(H-2^b) and DBA/2(H-2^d), was EAT-regressive, although the H-2 locus of B10 shifted to the haplotype d of DBA/2. DBA/2 itself is EAT-progressive. This indicates that at least H-2K and/or D loci as class I are not simply related to mouse strain susceptibility to EAT. B10.Y (H-2^\u003cpa\u003e) was EAT-regressive. B10.R III (H-2^r) was almost EAT-regressive with some individual variations. B10.BR (H-2^k) and B10.SM (H-2^v) were EAT-progressive. 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  1. 0 資料タイプ別
  2. 03 紀要論文
  1. 250 大学院医歯学総合研究科(医)
  2. 20 紀要
  3. 01 Acta medica et biologica
  4. Vol.47 No.1

Susceptibility of H-2 and Ly Congenic Mice to Ehrlich Ascites Tumors

http://hdl.handle.net/10191/1942
c52b332e-c7b9-42b7-b37b-294af1556bb8
プレビュー
名前 / ファイル ライセンス Actions
KJ00000007196.pdf KJ00000007196.pdf (2.3 MB)
item type 紀要論文 / Departmental Bulletin Paper(1)
公開日 2007-05-10
タイトル
タイトル Susceptibility of H-2 and Ly Congenic Mice to Ehrlich Ascites Tumors
タイトル
言語 en
タイトル Susceptibility of H-2 and Ly Congenic Mice to Ehrlich Ascites Tumors
言語
言語 eng
キーワード
主題Scheme Other
主題 H-2 congenic mice
キーワード
主題Scheme Other
主題 Ly congenic mice
キーワード
主題Scheme Other
主題 Ehrlich tumor
資源タイプ
資源 http://purl.org/coar/resource_type/c_6501
タイプ departmental bulletin paper
著者 SATO, Norimitsu L.

× SATO, Norimitsu L.

WEKO 52543

SATO, Norimitsu L.

Search repository
KATO, Akiko

× KATO, Akiko

WEKO 52544

KATO, Akiko

Search repository
FUJISAWA, Nobuyoshi

× FUJISAWA, Nobuyoshi

WEKO 52545

FUJISAWA, Nobuyoshi

Search repository
抄録
内容記述タイプ Abstract
内容記述 It has been shown that mice such as AKR/J (H-2^k), C3H/He (H-2^k), CBA/J (H-2^k), DBA/1 (H-2^q) and DBA/2 (H-2^d) are susceptible to Ehrlich ascites tumor (EAT), while other mice such as A/J (H-2^a), BALB/c (H-2^d), C57BL/6(H-2^b), C57BL/10(H-2^b), NZB/N(H-2^d) and SJL (H-2^s) are resistant to EAT. In addition, Ly haplotypes of the former EAT-progressive mouse strains classified were Ly-1^a (except AKR) and Ly-2^a, and those of the latter EAT-regressive mouse strains displayed Ly-1^b and Ly-2^b as common features. To see how the shift restricted in H-2 or in Ly haplotype affects their susceptibility, EAT progression in H-2 congenic and in Ly congenic mice was compared with that in standard mice. The results are as follows. 1) B10(H-2^b) as a standard was EAT-regressive. B10. D2(H-2^d), which is H-2 congenic between B10(H-2^b) and DBA/2(H-2^d), was EAT-regressive, although the H-2 locus of B10 shifted to the haplotype d of DBA/2. DBA/2 itself is EAT-progressive. This indicates that at least H-2K and/or D loci as class I are not simply related to mouse strain susceptibility to EAT. B10.Y (H-2^<pa>) was EAT-regressive. B10.R III (H-2^r) was almost EAT-regressive with some individual variations. B10.BR (H-2^k) and B10.SM (H-2^v) were EAT-progressive. In short, a shift in H-2 haplotype from b to k or b to v is significant in changing B10 susceptibility to EAT. 2) B6(H-2^b, Ly-1^b, Ly-2^b) as a standard was EAT-regressive. B6-Ly-1^a (H-2^b, Ly-1^a, Ly-2^b), B6-Ly-2^a (H-2^b, Ly-1^b, Ly-2^a) and B6-Ly-2^a, 3^a (H-2^b, Ly-1^b, Ly-2^a, Ly-3^a) were all EAT-regressive. No change in susceptibility was produced by the Ly-1,2,3,haplotype shift. Other B6 congenic mice such as B6-Tla^a (H-2K^k・D^b) and B6.C-H-2^<bm12>(H-2Aβ : bm12) were also EAT-regressive. From the results, it is concluded that H-2K, D loci including the TL region as class I, I-Aβ subregion as class II or Ly-1,2,3 loci, are not simply related to mouse strain susceptibility to EAT, suggesting the participation of non-MHC genes under H-2 associated control.
書誌情報 Acta medica et biologica
en : Acta medica et biologica

巻 47, 号 1, p. 21-25, 発行日 1999-03
出版者
出版者 Niigata University School of Medicine
ISSN
収録物識別子タイプ ISSN
収録物識別子 05677734
書誌レコードID
収録物識別子タイプ NCID
収録物識別子 AA00508361
著者版フラグ
値 publisher
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