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  1. 0 資料タイプ別
  2. 03 紀要論文
  1. 250 大学院医歯学総合研究科(医)
  2. 20 紀要
  3. 02 新潟医学会雑誌
  4. 第105巻第9号

4) 慢性骨髄増殖性疾患におけるvon Willebrand因子異常(新潟血栓止血研究会10周年記念特別例会記録)

http://hdl.handle.net/10191/39396
http://hdl.handle.net/10191/39396
09b7b28f-a759-4616-9d8c-22382b5c2802
名前 / ファイル ライセンス アクション
105(9)_628-631.pdf 105(9)_628-631.pdf (847.9 kB)
Item type 紀要論文 / Departmental Bulletin Paper(1)
公開日 2016-03-10
タイトル
タイトル 4) 慢性骨髄増殖性疾患におけるvon Willebrand因子異常(新潟血栓止血研究会10周年記念特別例会記録)
タイトル
言語 en
タイトル 4) 慢性骨髄増殖性疾患におけるvon Willebrand因子異常(新潟血栓止血研究会10周年記念特別例会記録)
言語
言語 jpn
キーワード
主題Scheme Other
主題 von Willebrand factor
キーワード
主題Scheme Other
主題 chronic myeloproliferative disorders
キーワード
主題Scheme Other
主題 in vivo proteolysis
キーワード
主題Scheme Other
主題 von Willebrand因子
キーワード
主題Scheme Other
主題 慢性骨髄増殖性疾患
キーワード
主題Scheme Other
主題 生体内蛋白分解
資源タイプ
資源 http://purl.org/coar/resource_type/c_6501
タイプ departmental bulletin paper
その他のタイトル
その他のタイトル Abnormalities of Plasma von Willebrand Factor in Patients with Chronic Myeloproliferative Disorders
著者 帯刀, 亘

× 帯刀, 亘

WEKO 130309

帯刀, 亘

Search repository
高橋, 芳右

× 高橋, 芳右

WEKO 130310

高橋, 芳右

Search repository
柴田, 昭

× 柴田, 昭

WEKO 130311

柴田, 昭

Search repository
著者別名
識別子 130312
識別子Scheme WEKO
姓名 Tatewaki, Wataru
著者別名
識別子 130313
識別子Scheme WEKO
姓名 Takahashi, Hoyu
著者別名
識別子 130314
識別子Scheme WEKO
姓名 Shibata, Akira
抄録
内容記述タイプ Abstract
内容記述 In chronic myeloproliferative disorders (CMPD) hemorrhagic complications are relatively common, in association with thrombocytosis. We studied the multimeric composition of plasma von Willebrand factor (vWf) in CMPD and investigated the possibilities of in vivo or ex vivo proteolysis. The relative amount of large multimers in patients with CMPD was significantly decreased in relation to platelet count as compared with normal subjects. It correlated negatively with platelet count, and positively with the ristocetin cofactor/vWf antigen ratio. All patients with primary myelofibrosis who had normal or subnormal platelet counts showed an almost normal multimeric pattern. The vWf abnormalities normalized with decreasing platelet counts after starting chemotherapy or when blast crisis occurred with decreased platelet counts. These abnormalities would be caused by in vivo proteolysis, because they were not corrected even when blood was obtained in the presence of protease inhibitors. The relative amount of large multimers of vWf did not correlate with plasma concentration of plasmin or elastase. Thus neither plasmin nor elastase appear to be the enzymes responsible for in vivo proteolysis.
書誌情報 新潟医学会雑誌
en : 新潟医学会雑誌

巻 105, 号 9, p. 628-631, 発行日 1991-09
出版者
出版者 新潟医学会
ISSN
収録物識別子タイプ ISSN
収録物識別子 00290440
書誌レコードID
収録物識別子タイプ NCID
収録物識別子 AN00182415
著者版フラグ
値 publisher
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