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The Cause of Hyperaminoacidemia in Completely Pancreatectomized Dogs
http://hdl.handle.net/10191/33491
http://hdl.handle.net/10191/33491e1fd18dd-d3cd-4bfe-91d8-1d69a37f833f
名前 / ファイル | ライセンス | アクション |
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38(4)_175-185.pdf (1.4 MB)
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Item type | 紀要論文 / Departmental Bulletin Paper(1) | |||||
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公開日 | 2015-10-01 | |||||
タイトル | ||||||
タイトル | The Cause of Hyperaminoacidemia in Completely Pancreatectomized Dogs | |||||
タイトル | ||||||
言語 | en | |||||
タイトル | The Cause of Hyperaminoacidemia in Completely Pancreatectomized Dogs | |||||
言語 | ||||||
言語 | eng | |||||
資源タイプ | ||||||
資源 | http://purl.org/coar/resource_type/c_6501 | |||||
タイプ | departmental bulletin paper | |||||
著者 |
Yoshikawa, Keiji
× Yoshikawa, Keiji× Mishina, Takeshi× Matsubara, Youichi× Muto, Terukazu |
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抄録 | ||||||
内容記述タイプ | Abstract | |||||
内容記述 | The rate of the hepatic glucose production was isotopically determined in combination with the measurement of plasma amino acid concentrations in completely pancreatectomized dogs. Marked hyperaminoacidemia mainly consisting of elevated glucogenic amino acids was observed regardless of the postoperative insulin replacement. Reduction of the elevated amino acids (except for the branched chain amino acids) by the exogenous glucagon was also observed. Particularly in cases where the plasma glucose level had been maintained at normal levels by the insulin, the decreasing of the plasma amino acid concentrations was associated with enhanced hepatic glucose production, indicating that the cause of hyperaminoacidemia in totally pancreatectomized dogs is inhibited gluconeogenesis from amino acids due to glucagon deficiency. | |||||
書誌情報 |
Acta medica et biologica en : Acta medica et biologica 巻 38, 号 4, p. 175-185, 発行日 1990-12 |
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出版者 | ||||||
出版者 | Niigata University School of Medicine | |||||
ISSN | ||||||
収録物識別子タイプ | ISSN | |||||
収録物識別子 | 05677734 | |||||
書誌レコードID | ||||||
収録物識別子タイプ | NCID | |||||
収録物識別子 | AA00508361 | |||||
著者版フラグ | ||||||
値 | publisher |